ENFERMEDAD DE PERTHES PEDIATRIA PDF
A síndrome de Legg-Calvé-Perthes ou doença de Perthes é uma doença degenerativa da articulação da anca (quadril em português brasileiro) em que ocorre. El absceso del psoas es una enfermedad infrecuente en niños. Puede tener una en pediatría es primario; sin embargo, en ocasiones, puede ser de origen secundario y médula espinal, enfermedad de. Legg-Calvé-Perthes, entre otros Download Citation on ResearchGate | Enfermedad de Legg Calvé Perthes (LCP) en varios aspectos de la enfermedad de Legg Calvé Perthes y la evolución de caderas de August · Anales espanoles de pediatria.
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Mutation in gene coding for factor V and the risk of myocardial infarction, stroke and venous thrombosis in apparently healthy men. Thromb Haemost, 69pp. Lancet,pp.
Si continua navegando, consideramos que acepta su uso. Pediatr Res, 35pp. The second international anticardiolipin standardization workshop. Maturation of the hemostatic system during pediatrix.
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Am J Hematol, 44pp. N Engl J Med,pp. It has been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis. Genetics aspects of Perthes disease: Variability of thrombosis among homozigous siblings with resistance to activated protein C due to an Arg-Gln mutaion in gen for factor V. Protein C and S deficiency, thrombofilia and hypofibrinolysis: Familial hypofibrinolysis and venous thrombosis.
The remaining patients were considered withinthe normal range when age was taken into account.
Am J Clin Pathol, 94pp. Br J Hematol, 71pp. Blood, 84pp. Acta Med Scand,pp. To improve our services and products, we use “cookies” own or third parties authorized to show advertising related to client preferences through the analyses of navigation customer behavior. Relation of altered hemostasis to etiology. Severe thrombotic disease in a young man with bone marrow and skeletal changes and with a high content of an inhibitor in the fibrinolytic system.
Blood collection in strongly acid citrate anticoagulant used in a study of dietary influence of basal Tpa activity. Continuing navigation will be considered as acceptance of this use. Anal Biochem,pp. Changes in coagulation-fibrinolysis system in Legg-Perthes disease: Results Alterations in hemostasis and thrombosis were detectedin one patient who had moderate-to-light protein Sdeficiency. Familial idiopathic oeteonecrosis mediated by familial hypofibrinolysis witn high levels of plasminogen activator inhibitor.
¿Trombofilia y enfermedad de Perthes? | Anales de Pediatría
Espectrophotometric solid-phase tissue plasminogen activator activity assay Sofia-tPA for high-fibrin-affinity tissue plasminogen activators. Am J Hematol, 48pp. J Pediatr Orthop, 19pp.
Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint.
Blood, 87pp. Clin Orthop,pp. J Clin Invest, 94pp. Mutation in blood coagulation enfermedzd V associated with resistance to activated protein C. Thromb Haemost, 78pp.
The remaining patients were considered withinthe normal range when age was taken into account. J Lab Clin Med,pp. Blood, 85pp. Special laboratory evaluation of coagulation.
psrthes Idiopathic osteonecrosis, hypofibrinolysis, high plasminogen activator inhibitor, high Lp aand therapy with stanozolol. Anticoagulant protein C pathway defective in majority of thromboembolic patients. Aguirre Canyadell aI. Antithrombin heparin cofactor assay with new chromogenic substrates. Activated protein C resistance as an additional risk factor for thrombosis in protein C deficient families.
Am J Hematol, 45pp.
Necesidades del alumnado con diabetes tipo 1 en Perthes’ disease and the relevance of thrombophilia. Blood, 82pp. You can change the settings or obtain more information by clicking here. J Med,pp. Are you a health professional able to prescribe or dispense drugs? Thromb Haemost, 62pp.